Abdominal aortic calcification progression and associated mortality in patients with end-stage renaldisease on hemodialysis

초록

Objective: Posterior reversible encephalopathy syndrome (PRES) is characterized by a clinical and radiological entity with rapid onset of seizures, headache, altered consciousness and visual disturbance with neuroimaging findings of reversible vasogenic subcortical edema. It is postulated to have a pathogenesis similar to hypertensive encephalopathy, although correlation with elevated blood pressure(BP) has not been demonstrated in chronic kidney disease(CKD).Despite the diverse initial trigger factors of PRES, uncontolled hypertension seems to be the common pathogenic pathway for PRES in CKD. For prevention of PRES in CKD patients, more intensive lowering of elevated BP should be initiated eagerly and BP should be well-maintained under control.